Formerly known as MOFAS: Minnesota Organization on Fetal Alcohol Syndrome
Exciting News from MOFAS
If you're looking for the Minnesota Organization on Fetal Alcohol Syndrome (MOFAS) you have come to the right place. We have some exciting news about our organization. We have a new name! MOFAS has officially been renamed Proof Alliance. Our mission remains the same: to prevent prenatal alcohol exposure and to improve the quality of life for people living with fetal alcohol spectrum disorders (FASD).
We now have the proof that prenatal alcohol exposure is a leading cause of brain injury in children. We have the proof that FASD is 100% preventable and people living with an FASD can reach 100% of their potential.
We seek to build powerful alliances with people with an FASD, their families, legislators, experts in the field, new partners, and community members to bring awareness, research, and services to this field.
Proof Alliance is rebranding, expanding, and we're moving! We have a new logo, website, and prevention campaign to help change the norms around drinking during pregnancy. And in May 2019 we will be moving to a stand-alone building. Proof Alliance commits to the people of Minnesota and we will continue to develop transformative programs to help Minnesotans impacted by FASD.
Preventing fetal alcohol spectrum disorders and supporting all impactedDonate
What is FASD?
Drinking alcohol during pregnancy can impact fetal development and cause irreversible birth defects and brain injury.1 Children with prenatal alcohol exposure are at risk of having fetal alcohol spectrum disorders (FASD). FASD is not a diagnosis but rather an umbrella term describing the range of birth defects caused by prenatal alcohol exposure. These effects may include physical, mental, behavioral, and/or learning disabilities with possible lifelong implications.2 There are many terms under the FASD umbrella, including these medical diagnoses:3
Fetal Alcohol Syndrome (FAS)
Alcohol Related Neuro-developmental Disorders (ARND)
Alcohol Related Birth Defects (ARBD)
Partial Fetal Alcohol Syndrome (pFAS)
The effects of prenatal alcohol exposure can impact each person differently. Some of the signs to look for that may indicate the need for an FASD assessment include:
The only known cause of FASD is prenatal alcohol exposure.15 FASD cannot be “passed down” from a parent with an FASD, and it cannot be caused by a partner’s drinking. FASD can be prevented by not drinking any alcohol throughout pregnancy, from conception to birth.16-20
Burd L, Blair J, Dropps K. Prenatal alcohol exposure, blood alcohol concentrations and alcohol elimination rates for the mother, fetus and newborn. Journal of Perinatology. 2012;32(9):652-659.
Noor S, Milligan ED. Lifelong Impacts of Moderate Prenatal Alcohol Exposure on Neuroimmune Function. Frontiers in Immunology. 2018.
Gross AC, Deling LA, Wozniak JR, Boys CJ. Objective measures of executive functioning are highly discrepant with parent-report in fetal alcohol spectrum disorders. Child Neuropsychology. 2015;2(4):531-538.
Stephen JM, Kodituwakku PW, Kodituwakku EL, Romero L, Peters AM, Sharadamma NM, Caprihan A, Coffman BA. Delays in auditory processing identified in preschool children with FASD. Alcoholism, Clinical and Experimental Research. 2012;36(10):1720-1727.
Vernescu RM, Adams RJ, Courage ML. Children with fetal alcohol spectrum disorder show an amblyopia-like pattern of vision deficit. Developmental Medicine and Child Neurology. 2012;54(6):557-562.
Glass L, Moore EM, Akshoomoff N, Jones KL, Riley EP, Mattson SN. Academic difficulties in children with prenatal alcohol exposure: Presence, profile, and neural correlates. Alcoholism: Clinical and Experimental Research. 2017;41(5):1024-1034.
Taggart TC, Simmons RW, Thomas JD, Riley EP. Children with Heavy Prenatal Alcohol Exposure Exhibit Atypical Gait Characteristics. Alcoholism: Clinical & Experimental Research. 2017;41(9):1648-1655.
Masotti P, Longstaffe S, Gammon H, Isbister J, Maxwell B, Hanlon-Dearman A. Integrating care for individuals with FASD: results from a multi-stakeholder symposium. BMC Health Services Research. 2015;15(1):1-12.
Young S, Absoud M, Blackburn C, Branney P, Colley B, Farrag E, & … Mukherjee R. Guidelines for identification and treatment of individuals with attention deficit/ hyperactivity disorder and associated fetal alcohol spectrum disorders based upon expert consensus. BMC Psychiatry. 2016; 16(1):324.
Flak AL, Su S, Bertrand J, Denny CH, Kesmodel US, Cogswell ME. The association of mild, moderate, and binge prenatal alcohol exposure and child neuropsychological outcomes: A Meta-analysis. Alcohol Clin Exp Res. 2014;38(1):214-26.
Terasaki LS, Schwarz JM. Impact of Prenatal and Subsequent Adult Alcohol Exposure on Pro-Inflammatory Cytokine Expression in Brain Regions Necessary for Simple Recognition Memory. Brain Sciences (2076-3425). 2017;7(10):1-16.
Panczakiewicz AL, Glass L, Coles CD, et al. Neurobehavioral deficits consistent across age and sex in youth with prenatal alcohol exposure. Alcoholism: Clinical and Experimental Research. 2016;40(9):1971-1981.
Pei J, Leung WSW, Jampolsky F, Alsbury B. Experiences in the Canadian criminal justice system for individuals with Fetal Alcohol Spectrum Disorders: Doublejeopardy?. Canadian Journal of Criminology & Criminal Justice. 2016;58(1):56-86.
Rangmar J, Dahlgren Sandberg A, Aronson M, Fahlke C. Cognitive and executive functions, social cognition and sense of coherence in adults with fetal alcohol syndrome. Nordic Journal of Psychiatry. 2015;69:472-478.
Fitzpatrick JP, Pestell CF. Neuropsychological Aspects of Prevention and Intervention for Fetal Alcohol Spectrum Disorders in Australia. Journal of Pediatric Neuropsychology. 2017;3(1):38-52.